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Referenz 224a Neurologie, 11. Auflage ; De Marinis M., Accornero N.: Recurrent neck pain as a variant of migraine: description of four cases. J. Neurol. Neurosurg. Psychiatry 62, 669-670 1997 ; . Department of Neurological Sciences, La Sapienza University, Rome, Italy. Four patients who had recurrent attacks of idiopathic unilateral neck pain and tenderness of the ipsilateral carotid artery are described. Two patients had never had headache. The other two had migraine without aura. All patients had dilatation of extracranial arteries during the attacks telethermographic examination ; , oculosympathetic hypofunction pupillary tests ; , and positive responses to vasoactive drugs which are commonly used for migraine treatment. Recurrent neck pain involving the carotid artery seems to be a variant form of migraine that may occur alone or in association with headache in patients with involvement of extracranial arteries.
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22. Grove, K. L., Sekhon, H. S., Brogan, R. S., Keller, J. A., Smith, M. S. & Spindel, E. R. 2001 ; Clin. Endocrinol. Metab. 86, 54205426. 23. Sekhon, H. S., Keller, J. A., Benowitz, N. L. & Spindel, E. R. 2001 ; Am. J. Respir. Crit. Care Med. 164, 989994. 24. Robinson, S. M. & Barker, D. J. 2002 ; Proc. Nutr. Soc. 61, 537542. 25. Khoo, M. C. 2000 ; Respir. Physiol. 122, 167182. 26. Hilaire, G. & Duron, B. 1999 ; Physiol. Rev. 79, 325360. 27. Shao, X. M. & Feldman, J. L. 2002 ; J. Neurophysiol. 88, 18511858. 28. Lechner, A. J. & Tull, D. S. 1986 ; Pediatr. Res. 20, 920924. 29. Fleming, P. J., Goncalves, A. L., Levine, M. R. & Woollard, S. 1984 ; J. Physiol. 347, 116. 30. Sawnani, H., Jackson, T., Murphy, T., Beckerman, R. & Simakajornboon, N. 2004 ; Am. J. Respir. Crit. Care Med. 169, 733738. 31. Oliff, H. S. & Gallardo, K. A. 1991 ; Front. Biosci. 4, D883D897. 32. Saint-Mleux, B., Eggermann, E., Bisetti, A., Bayer, L., Machard, D., Jones, B. E., Muhlethaler, M. & Serafin, M. 2004 ; J. Neurosci. 24, 6367. 33. Slotkin, T. A., Lappi, S. E., McCook, E. C., Lorber, B. A. & Seidler, F. J. 1995 ; Brain Res. Bull. 38, 6975. 34. Oncken, A., Henry, K. M., Campbell, W. A., Kuhn, C. M., Slotkin, T. A. & Kranzler, H. R. 2003 ; Pediatr. Res. 53, 119124. 35. Di Angelantonio, S., Matteoni, C., Fabbretti, E. & Nistri, A. 2003 ; J. Neurosci. 17, 23132322. 36. Changeux, J.-P. & Edelstein, S. J. 1998 ; Neuron 21, 959980. 37. Mansvelder, H. D., Keath, J. R. & McGehee, D. S. 2002 ; Neuron 33, 905919. 38. Cormier, A., Paas, Y., Zini, R., Tillement, J.-P., Lagrue, G., Changeux, J.-P. & Grailhe, R. 2004 ; Mol. Pharmacol. 66, 17121718. 39. Sparks, J. A. & Pauly, J. R. 1999 ; Psychopharmacology 141, 145153. 40. Cordero-Erausquin, M. & Changeux, J.-P. 2001 ; Proc. Natl. Acad. Sci. USA 98, 28032807. 41. Kinney, H. C., Filiano, J. J. & White, W. F. 2001 ; J. Neuropathol. Exp. Neurol. 60, 228247. 42. Weese-Mayer, D. E., Berry-Kravis, E. M., Zhou, L., Maher, B. S., Curran, M. E., Silvestri, J. M. & Marazita, M. L. 2004 ; Pediatr. Res. 56, 391395. 43. England, L. J., Levine, R. J., Mills, J. L., Klebanoff, M . A., Yu, K. F. & Cnattingius, S. 2003 ; Am. J. Obstet. Gynecol. 189, 939943. 44. Coleman, T., Britton, J. & Thornton, J. 2004 ; Brit. Med. J. 328, 965966 and cefzil, for instance, carisoprodol recreational.
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TABLE 12 Sensitivity, specificity and accuracy for SPECT from the three studies on PTCA Study Beygui, 200022 Hamasaki, 199627 Hecht, 199029 N 179 125 116 Stenosis % ; 50 Tracer Tl-201 Tl-201 Tl-201 Previous MI Yes No Yes Sensitivity 0.63 0.79 0.93 Specificity 0.77 0.78 0.77 Accuracy 0.71 0.78 0.86.
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Li Yao, Kagawa Medical University, Kagawa, Japan; Shinji Takai, Osaka Medical College, Takatsuki City, Japan; Yukiko Nagai, Takatomi Shokoji, Shoji Kimura, Hideyasu Kiyomoto, Masakazu Kohno, Kagawa Medical University, Kagawa, Japan; Kiyoshi Tamaki, Kurume University School of Medicine, Kurume, Japan; Mizuo Miyazaki, Osaka Medical College, Takatsuki City, Japan; Youichi Abe, Akira NISHIYAMA, Kagawa Medical University, Kagawa, Japan It is known that chymase is not involved in the angiotensin II formation in rats; however, recent studies also indicate that chymase contributes to tissue injury though non-angiotensin pathways, including the activation of transforming growth factor TGF ; -beta. In this study, we investigated the possible contributions of chymase to the activation of the TGF-beta Smad2 signaling pathway and renal injury in Dahl salt-sensitive DS ; hypertensive rats. Dahl salt-resistant DR ; and DS rats were randomized to receive a low salt L: 0.3% NaCl ; or high salt H: 8% NaCl ; diet for 4 weeks. In a separate group of DS H rats, a specific chymase inhibitor, 2- 5-formylamino-6-oxo-2-phenyl-1, 6-dihydropyrimidine-1-yl ; -N-[ -2-heptyl]acetamide NK3201: 10 mg kg day, p.o. ; was concurrently administered. mRNA expression of total TGF-beta1 and Smad2 activity in renal cortical tissues were measured by real-time PCR and Western blotting with an anti-phospo Smad2 antibody, respectively. DS H rats n 10 ; showed higher systolic blood pressure SBP: 205 -8 mmHg ; and urinary excretion of protein UproteinV: 336 -25 mg day ; than DS L rats 130 -4 mmHg and 58 -12 mg day, n 8 ; . Histologically, progressive sclerotic glomerular changes in DS H rats were accompanied by mast cell infiltration and immunopositive staining of TGF-beta and chymase. Compared with DS L rats, DS H rats showed increased renal cortical mRNA expression of TGF-beta1 2.8-fold ; and Smad2 activity 3.6-fold ; . On the other hand, there were no significant differences in these parameters among DR L n and DS L rats. In DS H rats, concurrent administration of NK3201 n 10 ; decreased SBP 165 -10 mmHg ; and UproteinV 144 -29 mg day ; , and markedly ameliorated glomerular injury. Furthermore, NK3201 decreased renal cortical mRNA expression of TGF-beta1 and normalized Smad2 activity in DS H rats. These data suggest that chymase plays a role, at least in part, in the development of renal injury in DS rats through the activation of the TGF-beta Smad2 signaling pathway.
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